The purpose of the Week 5 Disease Process Assignment Part 2 is for learners to:
- Develop professional presentation/communication skills.
- Link development of initial presenting signs and symptoms of disease to changes to tissue, organ and system functioning.
- Describe how the disease is diagnosed
- Describe pharmacological and non-pharmacological interventions for disease management.
Due Date: Sunday 11:59 PM MT at the end of Week 5
Course Outcomes
- Analyze pathophysiological mechanisms associated with select disease states.
- Relate research findings to the management of patients with complex pathophysiologic dysfunction.
Total Points Possible: 100 points
Requirements
Description of the Assignment
In Week 7, students will present an audio/video PowerPoint presentation using the Disease Process Presentation Guidelines and Rubric located under Course Resources. To ensure timely development of the Week 7 assignment and an opportunity to receive feedback from the course faculty, students will develop the Week 7 presentation in 2 parts. Parts 1 and 2 will be submitted as a PowerPoint presentation that contains thorough speaker notes. After students have received faculty feedback on Parts 1 and 2, students will develop the final presentation that is audio and video recorded and uploaded to the Week 7 Discussion Board.
Criteria for Content
- Link changes in the tissue, organ, and system functioning to the initial presenting signs and symptoms seen in primary careof the disease.
- Provide a brief description of how the disease is diagnosed.
- Provide a brief description of the pharmacological and non-pharmacological interventions used to treat and manage the disease.
- Summarizes the disease on final slide with concluding remarks; includes implication for nurse practitioner practice.
- Utilizes at least two current (within 5 years), peer-review scholarly sources to support presentation content.
- Reference slide and in-text citations depict references correctly cited according to APA.
Solution:
Diabetes mellitus type 1 (DM1), is a chronic health condition that occurs when the pancreas produces little or no insulin due to autoimmune beta cells in the pancreas (Katsarou et al., 2017). Insulin hormone allows the passage of sugar (glucose) from the bloodstream into the cells for energy production. DM1 is more prevalent in children and adolescents but can also develop in adults.
The digestive system breaks down carbohydrates into glucose which is then transported to the cells through the blood stream. The pancreas is responsible for secreting insulin hormone which allows for movement of glucose from blood to the body cells (Zaccardi, Webb, Yates, & Davies, 2016). Inside the cells, the glucose is burned along with oxygen in order to produce oxygen. For patients with type 1 diabetes, the pancreas does not make enough insulin to maintain blood glucose at normal levels (Katsarou et al., 2017). This increases glucose within the bloodstream and reduces glucose in cells for energy production. As such, the body tries to provide the cells with other fuel sources such as stored fats. The extensive burning of fat can produce by-products called ketones which are life-threatening in high amounts (Zaccardi et al., 2016)….Please click the Paypal icon below to purchase full solution for only $10